Search results “Differene between verapamil and diltiazem”
Calcium Channel Blockers - Nifedipine, Verapamil, Diltiazem & Amlodipine
http://usmlefasttrack.com/?p=431 voltage, L type channels, vascular smooth muscles, heart, hypertension, angina, prinzmetal's angina, raynauds, cardiac depression, dizziness, First Aid, for, USMLE, Step 1, wiki, define, wikipedia,
Views: 21286 USMLEFastTrack
Calcium Channel Blockers made Simple
I created this video with the YouTube Video Editor (http://www.youtube.com/editor)
Views: 80629 100lyric
Calcium Channel Blockers - For Nursing Students and Nurses!
EmpoweRN.com Here is the link for the additional NCLEX style questions! http://empowern.com/2015/02/calcium-channel-blockers/ Also, at the bottom of the page, there is a link to download the audio version of this video with & without music. Please also visit the references & recommended resources links... which you can find at the bottom of the page :) If this video has helped you in any way & you would like to see more videos like it: please give the video a "thumbs up" & also subscribe to the channel. I would like to thank the talented & intelligent contributes of this video: Rizalyn Joy Gadugdug Maria Salvacion Gonzales Yasmin Hashmi Artem Shestakov And Babar Hayatrana Disclaimer: These videos are intended for entertainment purposes only. Please follow the policy and procedures that your institution requires. Please note that the views, ideas & opinions expressed on this channel and in the videos on this channel are not necessarily of those of my employer or institution. The views expressed on this channel and in the videos channel do not represent medical advice. If you have specific medical concerns, please contact your physician. In order to protect patient privacy, all patient identifiers in all videos have been deleted or altered. The views expressed on this channel and in the videos on this channel are personal opinions. I am not an expert nor do I dispense medical advice or procedural specifications. The information I present is for general knowledge and entertainment purposes only. You need to refer to your own medical director, teachers and protocols for specific treatment information. It is your responsibility to know how best to treat your patient in your jurisdiction. Calcium Channel Blockers, as the name suggests, prevents or reduces the opening of these channels. There are different classes of Calcium channel blockers, but almost of all of them acts on the L-type voltage-gated calcium channel. Therefore, blocking or reducing calcium entry into these cells, means inhibiting calcium effects, and thus causes the following reaction: Vasodilation – by acting on vascular smooth muscle, Calcium channel blockers reduces contraction of the arteries which causes an increase in arterial diameter. This drugs primarily affect arteries, with minimal effects on venous vessels. Calcium Channel Blockers also have a Negative Inotropic effect – by acting on cardiac muscle, Calcium channel blockers therefore can reduce the force of the contraction in the heart. Calcium channel blockers also have a Negative Chrono-tropic – effect – by slowing down the conduction of electrical activity within the heart, therefore Calcium channel blockers may affect the rate of the heartbeat. They also can exhibit a Negative Dromo-tropic effect – by slowing down the conduction of the electrical activity of the heart, thus the conduction of velocity also decreases, particularly at the atrioventricular node. Which can ultimately slow the heart rate. Therapeutic Use Calcium Channel Blockers are used to control a variety of medical condition such as high blood pressure, chest pain, and tachyarrhythmia. As anti-hypertensive drug – The effects of calcium in the heart muscles, is that it causes the muscle to act aggressively, by contracting more forcefully. Calcium also stimulates vascular smooth muscle contraction resulting in narrow blood vessels. This series of events many times can result in high blood pressure. By preventing the entry of calcium into the heart muscle and vascular smooth muscles (particularly in the arteries), the heart muscle contraction will not be too strong and arterial vessels are able to relax and dilate, leading to lower blood pressure. As anti-anginal drug – The word angina is one that you will hear a lot as a nursing student and nurse. This is a fancy word for “chest pain.”
Views: 144346 EmpoweRN
Calcium channel blockers are usually well absorbed through the git which means that they go through first pass metabolism . However once entered into the blood stream calcium channel blockers are highly bound to plasma proteins, after doing their action calcium channel blockers are then metabolized by the liver and excreted renally.
Views: 17778 Pill Whiteboard
Support us on Patreon: https://www.patreon.com/speedpharmacology Follow us on Facebook: https://www.facebook.com/SpeedPharmacology/ Get Speed Pharmacology Merch Here: https://teespring.com/stores/speed-pharmacology **************************************************************************************************** Topics covered include: cardiac conduction system, SA node, AV node, bundle of His, bundle branches, purkinje fibers, cardiac action potential, pacemaker cells, conducting cells, contractile cells, mechanisms of arrhythmias, bradycardia, tachycardia, abnormal automaticity, triggered activity, Wolff–Parkinson–White syndrome, atrioventricular nodal reentry tachycardia, Vaughan-Williams classification, class I (A, B, C), class II, class III, class IV antiarrhythmic drugs. Antiarrhythmics mentioned include: Procainamide, Quinidine, Disopyramide, Lidocaine, Mexiletine, Flecainide, Propafenone, Propranolol, Metoprolol, Atenolol, Esmolol, Amiodarone, Dronedarone, Sotalol, Dofetilide, Ibutilide, Verapamil, Diltiazem, Digoxin, Adenosine, and Magnesium sulfate. Source of the animation of the cardiac conducting system: http://www.passmyexams.co.uk/GCSE/biology/cardiac-conduction-system.html
Views: 231462 Speed Pharmacology
Calcium Channel Blockers
In this video Dr. Mahmudul Hoque Jassy discusses about following topics 1. Classification of Calcium Channel Blocker 2. Indication of amlodipine , nifedipine SR, Larcanidipine, clinidipine and nimodipine 3. Mechanism of contration of vascular smooth muscle and role of voltage sensitive L-type calcium channel in contraction of vascular smooth muscle ( with details intracellular events). 4. How does Amlodipine / Nifedipine SR/ Larcanidipine/ Clinidipine lower BP? 5. Why Nifedipine is used in Raynaud phenomenon in COLD, in EMOTIONAL stress , in SLE and in Scleroderma. ( this indication is confirmed from professor Azizul kahhar sir) 6. Why CCB is used in preterm labor? What is the problem of preterm labour? 7. What is the role of nimodipine in Subaracnoid Hemorrhage and intracerebral hemorrhage with ventricular extension ( Many mechanisms are proposed in different books. but the most acceptable mechanism is discussed with approval of Professor Azizul Kahhar sir) 8. Common adverse effects of CCB( including ankle edema). 9. how Nifedipine and Felodipine produce reflex tachycardia? This video will be useful for medical and nursing students all over the world. Students who take preparation for USMLE, AMC, MRCP, MBBS may find this video helpful
Views: 9076 Pharmacology Videos
Support us on Patreon: https://www.patreon.com/speedpharmacology Follow us on Facebook: https://www.facebook.com/SpeedPharmacology/ Get Speed Pharmacology Merch Here: https://teespring.com/stores/speed-pharmacology **************************************************************************************************** Topics covered include: basic pathophysiology of hypertension, regulation of blood pressure, cardiac output, systemic vascular resistance, baroreceptors, alpha & beta receptors, vasoconstriction, vasodilation, renin-angiotensin-aldosterone system, bradykinin, nitric oxide. Mechanism of action of antihypertensive drugs and their side effects; adrenergic antagonists; alpha & beta blockers, centrally acting adrenergic agents, dihydropyridine & nondihydropyridine calcium channel blockers, loop, thiazide, potassium-sparing diuretics, renin inhibitors, angiotensin converting enzyme (ACE) inhibitors, angiotensin II receptor type 1 blockers (ARBs), endothelin receptor antagonist, dopamine-1 receptor agonist, peripheral vasodilators. Drugs mentioned include; Doxazosin, Prazosin, Clonidine, Methyldopa, Amlodipine, Felodipine, Nicardipine, Nifedipine, Diltiazem, Verapamil, Furosemide, Hydrochlorothiazide, Triamterene, Spironolactone, Aliskiren, Benazepril, Captopril, Enalapril, Lisinopril, Quinapril, Ramipril, Candesartan, Irbesartan, Losartan, Olmesartan, Valsartan, Bosentan, Fenoldopam, Sodium Nitroprusside, Nitroglycerin, Hydralazine, and Minoxidil.
Views: 196164 Speed Pharmacology
Amlodipine Nursing Considerations, Side Effects and Mechanism of Action Pharmacology for Nurses
Grab our free cheatsheet covering the 50 most commonly prescribed medications right here: http://www.nrsng.com/50meds View full post: https://www.nrsng.com/amlodipine-norvasc/ Listen to all the episodes at: https://www.nrsng.com/medmaster-podcast/ Amlodipine Generic Name: Amlodipine Trade Name: Norvasc Indication: hypertension, angina Action: blocks transport of calcium into muscle cells inhibiting excitation and contraction Therapeutic Class: antihypertensive Pharmacologic Class: Ca channel blocker Nursing Considerations  may cause gingival hyperplasia  grapefruit juice may increase drug level  monitor blood pressure and pulse prior to and during therapy  monitor intake and output  assess for signs of CHF  assess characteristics of angina  instruct patient of interventions for hypertension and how to take blood pressure
Diltiazem - New Medication Administration Set
A basic review of Diltiazem (Cardizem) and the new powdered administration set. Please keep comments constructive and respectful.
Views: 18053 BlueJFilms
Potassium channel blockers
Potassium Channel Blockers are easy! Check it out! I work hard to make and find the best videos for you to study from (it’s not easy)! You can help by suggesting any good videos you've seen in the comments below! Good luck in school!! Hey, guys. This is Dr. Joel with MedImmersion. You are watching a flash flood review series video where I review the highest yield stuff for your board exams in as little time as possible. In this video, I'm going to be talking about the potassium channel blockers, which are a class of the antiarrhythmic agents. I'll cover some general principles about the class as a whole, and then a few specific drugs in that category, or in this class. It's gonna be awesome, so stick around. If you watch a lot of MedImmersion videos, and they're helpful for you, think about getting an account at medimmersion.com. That will speed you up in your studying because when you're logged into MedImmersion on your browser and use the links in these videos, you will automatically skip past these lengthy introductions on my MedImmersion videos and also on other people's videos that I've linked to. And you'll be able to skip over a lot of the YouTube advertisements on my MedImmersion videos. Okay. Let's jump into it. This lecture is going to cover a subset of the antiarrhythmics, the potassium channel blockers. I'll give you an introduction as to what they are. Then, talk about some general principles that will include the mechanism of action, clinical uses, adverse or side effects, and then I will cover a couple of the highest yield examples in this class. Alright. First of all, if you need to review the entire topic of the antiarrhythmics, with a little bit of cardiac physiology, you should really go over and watch The Antiarrhythmic Agents Lecture first. This lecture, right now, is a little bit more focused, and I assume that you know a couple of things about antiarrhythmics. So, the potassium channel blockers, in the Vaughn Williams Antiarrhythmic Agent Classification, are the Class III antiarrhythmics. You should know that. And we use the potassium channel blockers and the Class I, or sodium channel blockers, for rhythm control. The Class IIs and Class IVs are more rate control. Four drugs that we're gonna talk about amiodarone, ibutilide, dofetilide, and sotalol, which you can remember by the mnemonic AIDS. As for the mechanism of action, these block myocardial potassium channels, and that has its primary effect on the specific potassium channels that are responsible for the delayed rectifier current, which have a very important contribution on the length of the action potential, and thus, the effective refractory period of cardiac myocytes. And to explain that a little bit further. Have you ever wondered why exactly the action potential of a neuron through the spinal cord or a peripheral neuron looks different than the action potential in myocardium. Well, it's built that way on purpose. The plateau phase, or the prolongation of the action potential, or the refractory period, gives cardiac tissue special properties that prevent it or contribute to prevention of arrhythmias. So, the picture on the left is what maybe a bland neuron action potential might look like in the peripheral nervous system. On the right, we have a cardiac action potential. And both of these are pretty bland images. They're not exactly right. But the point here is that there's a plateau where the cell stays in its non-polarized state for a little bit longer, for a period of time. Potassium plays a big part in that. The initial depolarization is caused by a rapid influx of sodium. And then, it's maintained in that depolarized state by both calcium and potassium trading places across the cell membrane in relatively small amounts. In phase three, which is the repolarization phase, finally there's a delayed switch, or a delayed rectifier current of potassium, which finally turns on and allows an efflux of positive ions, allowing the membrane potential to come back down to a very negative number. So, hopefully you can see why if we mess around with the potassium channels that contribute to phase three, we prolong or at least change the shape of the cardiac action potential, which, of course, would have an effect on some kinds of arrhythmias. So, it was pretty easy to see, I think, from that previous picture that delaying the potassium efflux during the repolarization phases increases or stretches out the action potential duration and also the effective refractory period. Also, these do not have any effect on the sodium channels, meaning that the conduction velocity, or phase zero, wouldn't be affected or decreased. And visually, it looks like this, the action potential stretched out and the effective refractory period is increased. Also, that means that the QT interval is prolonged. Okay. Does that make sense? We use the Class III antiarrhythmic agents for rhythm control.
Views: 6499 Med Immersion
Amlodipine Side Effects
This video is for the website http://www.amlodipinesideeffects.com. It is my testimonial of the use of the hypertension medication, Amlodipine. http://www.hangoutmarketingtrainingreview.net
Views: 38404 Brian Sloan
Antiarrhythmic Drugs
This is a brief overview of antiarrhythmic agents, or drugs used to resolve abnormal cardiac rhythms. I created this presentation with Google Slides. Image were created or taken from Wikimedia Commons I created this video with the YouTube Video Editor. ADDITIONAL TAGS Class IA antiarrhythmic agent Moderate sodium s, which s action potential duration Quinidine side effects blocks hERG , which results long QT and can cause torsades de pointes Procainamide Less prolongation QT segment, less TdP Disopyramide s force contraction heart Side effects: constipation, urinary retention, glaucoma IB antiarrhythmic Mild sodium s, which s action potential duration Lidocaine Intravenous only Mexiletine Can be administered orally IC antiarrhythmic Marked sodium s, doesn’t change action potential duration Flecainide Possibly produces an ventricular arrhythmias Propafenone Some beta er effects (bradycardia and cardiac inotropy) addition to changing AP duration by changing Na influx, Is also: phase 4 depolarization threshold potential sub degree Na+ AP duration change Beta-adrenergic receptor ers (beta ers) catecholamines (norepinephrine, epinephrine, dopamine) Reduces myocardial need for oxygen, can ischemia slope phase 4 depolarization s self-generated rhythmic firing heart (s automaticity) Prolong repolarization AV node → reentry Effectively s refractory period III antiarrhythmic s potassium s (delayed-rectifier potassium (DRK) s) Prolongs repolarization (phase 3) Amiodarone, Sotalol, Ibutilide, D etilide, Dronedarone III: Amiodarone Wide range effects through many mechanisms s sinus node firing s automaticity s reentrant circuits s Na, K, and Ca s ( I, III, IV antiarrhythmics) s alpha and beta ( II) adrenergic receptors → vasodilation and d intropy Treats many tachyarrhythmias: atrial flutter, atrial fibrillation, vtach, ventricular flutter, SVT Pharmacokinetically unique: absorbed slowly, deposits adipose tissue Half life 25-60 days → cannot easily diminish or reverse effects Side effects: pulmonary (pneumonia, pul fibrosis); cardiac (brady, arrhythmias, long QT, TdP); thyroid (due to iodine); GI; CNS Amiodarone Wide range effects through many mechanisms sinus node firing; s automaticity; s reentrant circuits; Na, K, and Ca alpha and beta adrenergic receptors vasodilation and intropy Treats many tachyarrhythmias: atrial flutter, atrial fibrillation, vtach, ventricular flutter, SVT Pharmacokinetically unique: absorbed slowly, deposits adipose tissue Half life 25-60 days Side effects: pulmonary (pneumonia, pul fibrosis); cardiac (brady, arrhythmias, long QT, TdP); thyroid (due to iodine); GI; CNS High rates torsades de pointes Dronedarone (amiodarone analog without iodine) Gastrointestinal side effects but not TdP Sotalol Calcium L-type Ca2+ Most effective cells dependant on Ca (SA, AV nodes) transmission through AV node (for rapid atrial pulses) Terminates reentrant rhythms Treats AV nodal reentrant tachycardia (primary treatment) Side effects: hypotension and heart failure pts taking beta-ers Diltiazem and Verapamil Digoxin Inhibits activity sodium potassium pump (Na+-K+ ATPase inhibitor) Treats heart failure complicated with atrial fibrillation (by decreasing heart rate) s vagal tone; reduces sympa tic activity Opens potassium (K+ activator) Intravenously with saline flush (short 10 s half life) Hyperpolarizes cells Allows for rapid termination reentrant supraventricular tachycardia chemical defibrillator
Views: 80665 MedLecturesMadeEasy
Confused about DILTIAZEM Formulations?
Learn different formulation about diltiazem hydrochloride
What is CARDIOVERSION? What does CARDIOVERSION mean? CARDIOVERSION meaning, definition & explanation
What is CARDIOVERSION? What does CARDIOVERSION mean? CARDIOVERSION meaning - CARDIOVERSION pronunciation - CARDIOVERSION definition - CARDIOVERSION explanation - How to pronounce CARDIOVERSION? Source: Wikipedia.org article, adapted under https://creativecommons.org/licenses/by-sa/3.0/ license. Cardioversion is a medical procedure by which an abnormally fast heart rate (tachycardia) or other cardiac arrhythmia is converted to a normal rhythm using electricity or drugs. Synchronized electrical cardioversion uses a therapeutic dose of electric current to the heart at a specific moment in the cardiac cycle, restoring the activity of the electrical conduction system of the heart. (Defibrillation uses a therapeutic dose of electric current to the heart at a random moment in the cardiac cycle, and is the most effective resuscitation measure for cardiac arrest associated with ventricular fibrillation and pulseless ventricular tachycardia.) Pharmacologic cardioversion, also called chemical cardioversion, uses antiarrhythmia medication instead of an electrical shock. To perform synchronized electrical cardioversion, two electrode pads are used (or, alternatively, the traditional hand-held "paddles"), each comprising a metallic plate which is faced with a saline based conductive gel. The pads are placed on the chest of the patient, or one is placed on the chest and one on the back. These are connected by cables to a machine which has the combined functions of an ECG display screen and the electrical function of a defibrillator. A synchronizing function (either manually operated or automatic) allows the cardioverter to deliver a reversion shock, by way of the pads, of a selected amount of electric current over a predefined number of milliseconds at the optimal moment in the cardiac cycle which corresponds to the R wave of the QRS complex on the ECG. Timing the shock to the R wave prevents the delivery of the shock during the vulnerable period (or relative refractory period) of the cardiac cycle, which could induce ventricular fibrillation. If the patient is conscious, various drugs are often used to help sedate the patient and make the procedure more tolerable. However, if the patient is hemodynamically unstable or unconscious, the shock is given immediately upon confirmation of the arrhythmia. When synchronized electrical cardioversion is performed as an elective procedure, the shocks can be performed in conjunction with drug therapy until sinus rhythm is attained. After the procedure, the patient is monitored to ensure stability of the sinus rhythm. Synchronized electrical cardioversion is used to treat hemodynamically unstable supraventricular (or narrow complex) tachycardias, including atrial fibrillation and atrial flutter. It is also used in the emergent treatment of wide complex tachycardias, including ventricular tachycardia, when a pulse is present. Pulseless ventricular tachycardia and ventricular fibrillation are treated with unsynchronized shocks referred to as defibrillation. Electrical therapy is inappropriate for sinus tachycardia, which should always be a part of the differential diagnosis. Various antiarrhythmic agents can be used to return the heart to normal sinus rhythm. Pharmacological cardioversion is an especially good option in patients with fibrillation of recent onset. Drugs that are effective at maintaining normal rhythm after electric cardioversion, can also be used for pharmacological cardioversion. Drugs like amiodarone, diltiazem, verapamil and metoprolol are frequently given before cardioversion to decrease the heart rate, stabilize the patient and increase the chance that cardioversion is successful. There are various classes of agents that are most effective for pharmacological cardioversion.
Views: 2704 The Audiopedia
Calcium Channel Blockers
-- Created using PowToon -- Free sign up at http://www.powtoon.com/youtube/ -- Create animated videos and animated presentations for free. PowToon is a free tool that allows you to develop cool animated clips and animated presentations for your website, office meeting, sales pitch, nonprofit fundraiser, product launch, video resume, or anything else you could use an animated explainer video. PowToon's animation templates help you create animated presentations and animated explainer videos from scratch. Anyone can produce awesome animations quickly with PowToon, without the cost or hassle other professional animation services require.
Views: 25118 Alison Fowler
Calcium channel blocker
Calcium channel blockers (CCB), calcium channel antagonists or calcium antagonists are a number of medications that disrupts the movement of calcium (Ca2+) through calcium channels. Calcium channel blockers are used as antihypertensive drugs, i.e. as medications to decrease blood pressure in patients with hypertension. CCBs are particularly effective against large vessel stiffness, one of the common causes of elevated systolic blood pressure in elderly patients. Calcium channel blockers are also frequently used to alter heart rate, to prevent cerebral vasospasm, and to reduce chest pain caused by angina pectoris. Despite their effectiveness, CCB's often have a high mortality rate over extended periods of use, and have been known to have multiple side effects. Potential major risks however were mainly found to be associated with short-acting CCBs. This video is targeted to blind users. Attribution: Article text available under CC-BY-SA Creative Commons image source in video
Views: 23655 Audiopedia
Hypertrophic cardiomyopathy (HCM) - causes, symptoms, treatment & pathology
What is hypertrophic cardiomyopathy (HCM)? HCM is a type of cardiomyopathy where the cardiac muscles grow, which ultimately leads to poor filling and diastolic heart failure. Find more videos at http://osms.it/more. Find more videos at http://osms.it/more. Study better with Osmosis Prime. Retain more of what you’re learning, gain a deeper understanding of key concepts, and feel more prepared for your courses and exams. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways and more when you follow us on social: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Thank you to our Patreon supporters: Sumant Nanduri Omar Berrios Alex Wright Suzanne Peek Arfan Azam Mingli Féng Osmosis Vision: Empowering the world’s caregivers with the best learning experience possible.
Views: 15808 Osmosis
The Treatment Of High Blood Pressure II
As was discussed in a previous vignette, there are several types of blood pressure medications to lower high blood pressure. Diuretics, ACE inhibitors and beta blockers were all discussed. All have a role to play, and all attack high blood pressure at different places in the body’s process of maintaining blood pressure. Often, combinations must be used to control high blood pressure. This vignette will further discuss different types of medications that are available to control high blood pressure. The next kind of medicine to control high blood pressure is the calcium channel blockers. They are further divided into two different groups: one that opens up the arteries of that lowering the blood pressure inside such as nifedipine and amlodipine, and one that lowers the heart rate such as Verapamil or Diltiazem. These medications are not really considered first-line therapies, but at times they do have a role to play in lowering blood pressure. Verapamil and Diltiazem are also used in certain heart rhythms when the heart rhythm is too fast. The good news with these medications is that there is increasing evidence that they may reduce stroke and heart attacks. However, they do have side effects including headache and swelling in the legs, and of course there’s always a chance that the heart rate can be lowered too much, causing heart failure or just general fatigue. Angiotensin II receptor blockers (ARBs) are a new kind of blood pressure medicine that are being used more and more. They work in a similar way to the ACE inhibitors. They differ in that they do not cause a cough, and so many physicians use this type of medication when the ACE inhibitors cause a cough. This kind of medication includes Losartan, Olmesartan, Eprosartan, Telmisartan, Candesartan, Valsartan, and Irbesartan. It is now thought that they have similar effects on heart failure, kidney disease and heart attacks that ACE inhibitors have, although there are not as many studies done on ARBs as there are on the ACE inhibitors. On a more personal note, I very rarely use these medications because of their high costs and to me personally, they just don’t seem very strong in actually reducing high blood pressure. There are several other classes of medication that are used less frequently. For instance, in very high blood pressures, some doctors use a medication called hydralazine that helps open the arteries up a bit allowing the pressure inside the artery to decrease. This medication is used three times a day, and often causes lots of swelling in the legs. Furthermore, not everyone responds to it. There is also a class of high blood pressure medicines called alpha blockers, such as doxazosin, that actually increase the risk of stroke and heart failure when used. Finally, there probably needs to be a special word given to the medication called clonidine. Clonidine or Catapress is available as a pill or a patch. It’s considered a pretty strong medication, and is one that used a lot when blood pressures are very high, especially if we think somebody is at or near a stroke or kidney failure. It is not used very often long-term because of its two main side effects: it’s very sedating, and it gives a very dry mouth. Both of these symptoms get a little better over time, but for most people they never completely get used to them, either. As one can see, there are a lot of choices out there for high blood pressure, and this vignette just scratches the surface. If you have high blood pressure, please consult your physician as to what to do, but we hope that the information above gives you a little bit more information as to how we approach and treat high blood pressure.
Views: 4635 ChristianWebMD.com
Dihydropyridine & Ryanodine Channels - Excitation/Contraction Coupling
How is calcium released to cause muscle contraction? This question is answered in this video. The dihydropyridine receptors and ryanodine receptor channels are discussed.
Views: 3603 PhysioPathoPharmaco
Calcium Channel Blockers
Learn why calcium channel blockers are prescribed and understand the different classes, the fun way. - created at http://goanimate.com/
Views: 150 Cheri927
Antiarrhythmic Drug Classes
Learning the Anti-Arrhythmic Agents just got a whole lot easier! ***MedImmersion to the rescue*** Listen guys, I really hope this video helps you in school. I definitely had fun making it! If you have questions, comments, or even criticisms...please, leave a comment. I love teaching and working with students, so your comments mean a lot to me! Good Luck in school! Hey YouTube, this is Dr. Joel. In this video, I'm gonna be covering the antiarrhythmic agents. I'm gonna start with a review of cardiac physiology, and then jump right into the agents themselves. I'll cover the Class I, Class II, Class III, Class IV, Class V, and then just give you some departing thoughts and then I will finish off with a couple of knowledge challenge questions, just to see where you're at. OK? Let's get started. In order to do a really good review of the cardiac antiarrhythmic agents, it's first important important for me to cover a little bit of cardiac physiology, starting first with the cardiac action potential. And that's because this action potential is a little bit different than the action potential that you're going to see in nerves. Also, a solid understanding of this action potential will help you later understand why the drugs work the way they do. So, this picture on the right represents a cardiac action potential. And, one thing that you need to understand is that this action potential is going to be a little bit different depending on which part of the heart you're measuring. However, the principles that I'm about to cover will apply to all of those tissues in the heart. And, if you want to, you can click on this link, which will take you to a picture that I think does a really cool job about showing the differences in the cardiac action potential in the different sections of the heart and that also how all those electrical depolarizations add up to make the electrocardiogram wave form. Anyway, on the X axis, we have time and on the Y axis, we have voltage. In the polarized state, the heart rests at about negative 95 millivolts. An action potential cycle takes about 200 milliseconds. And that number changes depending on which part of the heart you're in or which tissue you're sampling. So, on this graph, you can see that the heart starts at about negative 95 millivolts then it very quickly shoots up to about 20 or so, by this graph, pause at 20 millivolts. It stays there for a bit, and then the cell starts to repolarize itself. And that's the cycle. I'm going to add a cell membrane at the top of this picture and I'm going to walk through the phases of the action potential one at a time and what I want you to do is, I want you to imagine that above this cell membrane is the extracellular space and below this membrane is the intracellular space. OK, starting off with Phase 0, which is the depolarization phase. This is caused by a opening of voltage-gated sodium channels. And these are very fast, rapid-acting channels that allow a large amount of sodium to move very quickly. Sodium is positively charged, so if positive things come into the cell, then the cell becomes more positive. OK, does that make sense? Basically, that's why you see this huge skyrocketing here of the voltage from negative 95 to positive 20. It's because those positive sodium ions are moving in very quickly. Next is Phase 1, which is the initial repolarization phase, which is basically caused by the rapid inactivation of those sodium channels. Almost as quickly as they open, they start to close again. At the same time, voltage-gated potassium channels start to open allowing potassium to efflux or exit the cell. Potassium is also positively charged. So if you have positive things leaving the cell, then the cell becomes more negative, right? And that's why there's a little dip there in the voltage. Next, with Phase 2, you get calcium channels and they begin to open. Calcium, again, also positive. Positive things coming into the cell would make the cell more positive. But potassium is still moving out, so that would make the cell more negative, and hence you get this plateau phase. It kind of balances out for a little bit. It's not exactly flat, but it's close. We still call it the plateau phase. And, as you know, the calcium plays an effect on how the muscle cells contract. So that's important as well for contraction. Next is the rapid repolarization phase, which is Phase 3. More of the voltage-gated slow potassium channels are opening and they allow more potassium to rush out and the calcium channels begin to close so the cell starts to move back down to a negative value, a strong negative value. And you have to remember, the sodium-potassium ATPase pump is also chugging along this whole time. It's still working, it's still pumping potassium in and sodium out, which is just another factor that is driving that cell back down to its polarized state. Lastly is the fourth phase, which is the resting potential phase.
Views: 77692 Med Immersion
Medications for high blood pressure (Calcium channel blockers) interacts with FORSKOLIN Forskolin might decrease blood pressure. Taking forskolin with medication for high blood pressure might cause your blood pressure to go too low. Some medications for high blood pressure include nifedipine (Adalat, Procardia), verapamil (Calan, Isoptin, Verelan), diltiazem (Cardizem), isradipine (DynaCirc), felodipine (Plendil), amlodipine (Norvasc), and others. Medications that increase blood flow to the heart (Nitrates) interacts with FORSKOLIN Forskolin increases blood flow. Taking forskolin with medications that increase blood flow to the heart might increase the chance of dizziness and lightheadedness. Some of these medications that increase blood flow to the heart include nitroglycerin (Nitro-Bid, Nitro-Dur, Nitrostat) and isosorbide (Imdur, Isordil, Sorbitrate). http://www.youtube.com/watch?v=4WI5I6a1E3c http://www.youtube.com/user/MultiHerbalHealth
Views: 5349 Herbal Health
Cardizem Video
We were faced with the following challenge. Cardizem was added as an optional medication for the region. We opted to use it because of its apparent benefits over Lopressor and the fact that it can be used standing order. The issue is that the only cost effective preparation requires some specific education regarding its use. We felt that there needed to be training beyond what was covered in the CNYEMS rollout before we put this medication on the rigs. We did not have time to put everyone through face-to-face training so we are trying this different approach. We made a video. A bunch of us (who would probably have preferred to remain anonymous if you didn't already know who we are) made the above video. Rescue 911, for those of you who don't recall, was one of the original "reality" TV shows that began its original run in 1989. It is known for being hosted by William Shatner and including rather melodramatic clips of personal interviews during the recreation of each incident. The first part of the video is modeled after the show. Following this are director's comments reminiscent of a late night infomercial. We even have an appearance by our own Air-1 pilot, Sergeant "Lieutenant" Jim. Finally, I would like your feedback. We are implementing training ideas on a trial and error basis (a highly heuristic method, if you will). So far we have received excellent feedback on the rig check reviews. Feedback is imperative to make sure that we provide you with material that is worthwhile and meeting your needs. Special thanks goes out to everyone who participated. I'd especially like to recognize folks behind the scenes including Dan who spent a bunch of time shooting footage, editing and adding effects and my partner in training (and cinematic producer / director) Susie. Thank you. --Dr. Fullagar WAVES Member Specific Information: The first portion of the video contains a number of answers, if you pay close attention, to the quiz you were e-mailed. The second portion has somewhat more direct answers to questions with a demonstration on how to mix and draw up the diltiazem properly. WAVES ALS Providers: Please complete and return the quiz you were e-mailed to Susie. The quiz helps us document that you have an understanding of the content that was trying to be conveyed.
Views: 723 WAVESAmbulance
What does prazosin mean?
What does prazosin mean? A spoken definition of prazosin. Intro Sound: Typewriter - Tamskp Licensed under CC:BA 3.0 Outro Music: Groove Groove - Kevin MacLeod (incompetech.com) Licensed under CC:BA 3.0 Intro/Outro Photo: The best days are not planned - Marcus Hansson Licensed under CC-BY-2.0 Book Image: Open Book template PSD - DougitDesign Licensed under CC:BA 3.0 Text derived from: http://en.wiktionary.org/wiki/prazosin
SVT or Supraventricular Tachycardia
This video demonstrates the treatment of a patient with supraventricular tachycardia (SVT) using adenosine.
Views: 160092 Larry Mellick
What is the mechanism of amlodipine?
Pharmacology: Amlodipine is a second-generation calcium channel blocker.
Difference between the Coronary Calcium Score and the Carotid Intima Media Thickness scan
Dr Backs discusses the difference between the Coronary Calcium Score and the Carotid Intima Media Thickness scan, two testing options available for patients to define their arterial health!
Views: 225 Craig Backs
What are the risks or side effects of medications used to manage arrhythmia? (Evgueni Fayn, MD)
Evgueni Fayn, MD, Medical College of Wisconsin electrophysiologist, talks about the risk factors and side effects of drugs used to treat arrhythmia or irregular heartbeat. Dr. Fayn is part of the Arrhythmia Program team at Froedtert & The Medical College of Wisconsin. http://www.froedtert.com/arrhythmia
Specific Antidotes for Drug Overdose/Poisoning
Drug overdose & Antidotes are enumerated in a simple way for revision. Enjoy & Learn ! ______________________________________________________________ Please Visit & Subscribe Our Channel for Latest Videos: https://www.youtube.com/lastsecondmedicine Visit us on Facebook: https://www.facebook.com/lastsecondmedicine Follow us on Twitter: https://twitter.com/Last_Second_Med Visit Blog www.lastsecondmedicine.blogspot.com Support us on Patreon: https://www.patreon.com/lastsecondmedicine Please Leave you valued suggestion in Comments.
Views: 2587 Last Second Medicine
Antiarrhytmics (Lesson 7 - How to Choose the Right Med and Classic Pitfalls)
A discussion of how to choose the right antiarrhythmic to rate control and pharmacologically cardiovert atrial fibrillation and atrial flutter, as well as converting and prevention of ventricular tachycardia. An overall summary of this series is also presented.
Views: 6516 Strong Medicine
How do antiarrhythmic drugs work ?  | Health FAQS for Better Life
Antiarrhythmics (video) heart and stroke foundationdefinition of antiarrhythmic drugs by medical medications. Neither anti arrhythmic drugs nor catheter ablation decrease the risk of stroke these treatments are designed to control work well for some people, but not all. What does antiarrhythmic drugs mean? Antiarrhythmic work best when they are at constant levels in the blood. The ultimate goal of antiarrhythmic drug therapy is to restore normal rhythm and conduction. 7 2003 antiarrhythmic drug pharmacology. Do antiarrhythmic drugs work? Some reflections on the implications anti arrhythmic patient uk. 13 recognize and categorize different antiarrhythmic drugs by their mechanism of how do class i drugs exert their antiarrhythmic effects? . Symptomatic tachycardias and premature beats be treated with a variety of antiarrhythmic drugs. Antiarrhythmics texas heart institute information center cv pharmacology. Antiarrhythmic drugs are used to decrease or increase conduction velocity class ii antiarrhythmic medicines beta blockers, which work by blocking the impulses that cause an irregular heart rhythm and interfering with hormonal influences (such as adrenaline) on heart's cells. To help keep levels learn more about types of antiarrhythmic medications from osu heart and they must be tailored to each individual's specific needs do have side effects start studying drugs. They be used to why would i prescribed an anti arrhythmic? Calcium channel blockers such as verapamil and diltiazem work in a similar way. When it is not possible to revert normal sinus rhythm, drugs be used prevent more serious and possibly lethal arrhythmias from occurring. How do the pros and cons compare for now? . Medications for arrhythmia american heart association. By doing this, they also reduce blood pressure and heart rate 14 when taken exactly as prescribed, medications can do wonders. Mechanisms of action antiarrhythmic drugs springer. Antiarrhythmic drugs cvpharmacology antiarrhy antiarrhythmic url? Q webcache. Antiarrhythmics texas heart institute information centerantiarrhythmics center. Digoxin your doctor has determined that you have an abnormal heart rhythm would be best we're working on a new format for chapters, and like opinion. It is, therefore, not a true classification in that it does aggregate drugs into categories most antiarrhythmic are grouped 4 main classes (vaughan williams conduction fast channel tissues (working atrial and ventricular myocytes, be prescribed for people with tachycardia (fast these medications work to correct the rhythm of heart. Digoxin does not fit into this classification 6 as the number of available drugs with antiarrhythmic properties increased, need for a author ethan levine, domost notably by arrhythmia working group european society cardiology, who, agents, also known cardiac dysrhythmia medications, are class v agents work other or unknown mechanisms. Antiarrhythmic drugs are grouped in classes according to how they work 1if your heart beats too fast, antiarrhythmic be prescribed. New insights into how antiarrhythmic drugs work phys. Intro_to_antiarrhythmics [tusom. Medicines not work unless taken as prescribed anti arrhythmic drugs are designed to treat an abnormality of the heart rhythm. Googleusercontent search. Pharmwiki] tmedwebantiarrythmic drugs astate. It also makes no allowance for the fact that some medications work but because it slows recovery of sodium channels from inactivation does not shorten (and to understand how antiarrhythmic drugs work, need which decrease incidence va's do increase) risk 29 new insights into function numerous membrane proteins, with clear mechanism so your first doses are given you in hospital we can why i have go receive this medication? Cleveland clinic will insurance company preauthorize stay. Antiarrhythmic drugs have been used as an effective measure to treat or prevent accumulated studies on the mechanism of antiarrhythmic agents, however, despite major advances in our understanding mechanisms cardiac arrhythmias and how appear work, there remains much with anti arrhythmic heart beat too slowly (bradycardia), quickly (tachycardia) irregularly. Ablation for atrial fibrillation antiarrhythmic drug pharmacology. Drug cabinet anti arrhythmics british heart foundation. You should know how these drugs interact with each other 22 3 if rate control doesn't work, then a rhythm medication be used to try for more information, see what do we about multaq 1 'antiarrhythmic drugs', american heart association, copyright, 2006 anti arrhythmic agents work by blocking the sodium channel, potassium in order achieve rapid therapeutic levels, drug loaded blood thinners reduce but not totally eliminate risk of stroke. Ohio state conditions and treatmentspros cons of medication vs. What is a good mnemonic to remember examples of anti arrhythmic drugs? How do class ii antiarrhythmics work? . This is a here's how they work depending on the drug, do this in one of tw
Views: 177 BEST HEALTH Answers
UAB trial to test promising new treatment approach for diabetes
Anath Shalev, MD, director of the UAB Comprehensive Diabetes Center, explains the “repurposing of verapamil as a beta cell survival therapy in type 1 diabetes” trial, which will test an approach different from any current diabetes treatment. The trial is based on Shalev's research showing that verapamil, a common blood pressure drug, reverses diabetes in animal models. Learn more about the trial, and the research behind it, here: http://www.uab.edu/news/innovation/item/5508-in-human-clinical-trial-uab-to-test-drug-shown-to-completely-reverse-diabetes-in-human-islets-mice Follow the trail of discoveries that led to the new trial here: http://themixuab.blogspot.com/2014/11/discovery-route-path-to-potential.html?+news
Acid Reflux / GERD
Acid Reflux / GERD Physiology
Support us on Patreon: https://www.patreon.com/speedpharmacology Follow us on Facebook: https://www.facebook.com/SpeedPharmacology/ Get Speed Pharmacology Merch Here: https://teespring.com/stores/speed-pharmacology **************************************************************************************************** Topics covered include: absorption (passive diffusion, facilitated diffusion, active transport, endocytosis), distribution (volume), metabolism (phase I, phase II), elimination (zero, first-order kinetics), steady state, cytochrome P450 (inducers, inhibitors)
Views: 273273 Speed Pharmacology
Paraneoplastic Syndrome - high levels of PTHrH, Calcitriol, Erythropoietin & Lambert Eaton Syndrome
http://usmlefasttrack.com/?p=5360 Paraneoplastic, Syndrome, -, Increased, levels, of, PTHrH,, Calcitriol,, Erythropoietin, &, Lambert, Eaton, Syndrome, Findings, symptoms, findings, causes, mnemonics, review, what is, video, study, Rapid Review, Clinical presenation, First Aid, for, USMLE, Step 1, images, wiki, define, wikipedia, 2013, videos, exam, prep, easy, What is usmle, mnemonic, causes,
Views: 3340 USMLEFastTrack
Beta Blocker Side Effects
For education about preventing heart attacks, visit my website http://CardioGauge.com Please see my new channel for videos about specific Beta Blockers: Metoprolol, Carvedilol, Bisoprolol, Atenolol, Propranolol, Nadolol. https://www.youtube.com/channel/UC_z1l_iS5ZJFPi2FMHED95g Thanks!
Views: 54848 nathan ritter
003 - PreRegCentral: Hypertension and its Management Part 2
This video briefly outlines the key points of the role of Calcium channel blockers and Diuretics in the management of Hypertension Please subscribe to our channel and follow us on Facebook and Twitter for regular updates: Facebook: https://www.facebook.com/prereg.central Twitter: https://twitter.com/PreRegCentral All content (visual and audio) was created by PreReg Central
Views: 648 Pre Reg Central
(CC) Top 200 Drugs Chapter 6 Cardio Nursing Pharmacology by Suffix (Memorizing Pharmacology)
FREE Audiobook: https://www.amazon.com/Memorizing-Pharmacology-A-Relaxed-Approach/dp/B01FSR7XZO/ Allnurses.com article: http://allnurses.com/nursing-student-assistance/memorizing-pharmacology-video-1111129.html Website: https://www.memorizingpharmacology.com/ Top 200 Drugs Pronunciation and Mnemonics and review of prefixes, infixes, and suffixes of medications to go with book Memorizing Pharmacology: A Relaxed Approach / How to survive nursing school pharmacology
Views: 27134 Tony PharmD
LFT patterns and causes of jaundice
Check out my other videos: - Introduction to liver function tests https://youtu.be/QSviMc1mvuU - Introduction to Jaundice https://youtu.be/IwOhTefAKzo Happy learning!
Views: 1847 Jaz Singh
define CALCIUM
Tulip is a female droid, at Your Service.
Norvasc (calcium channel blocker) [FOM Productions]
A commercial for a calcium channel blocker Made by Foundations of Medicine Students at CAPS All rights Reserved to R. Kelly
Views: 907 Asad Morani
Nitrovasodilators and Angina Pectoris Part 3
DescriptionIn this video we discuss the mechanism of action of nitrovasodilators and consider why they can be used to treat Angina Pectoris
Views: 597 Ben1994
Questions & Answers 216
In This Video: 08:04- Gavin -- My question is that for about 8 years now I have been suffering of folliculitis of the scalp. What began as small pimples in anisolated area, eventually over time spread to pretty much the whole crown of my head, and now I have a raised red scar, which is apparently due to the destruction of the hair follicle, and I'm not beginning to see hair loss around the crown of my head. 20:14- Christa -- I have gone through all 12 Detox kits, and I am still taking Stomach & Bowels #3, Kidneys, Lymphatic, Adrenal, Parasite, Brain and Nerve and many others since June 2013. I am mainly on fruits and berries and salads, steamed vegetables, and some soaked nuts in the evening. I am very thin and can't eat less. I do get so hungry at times. I live in cold Canada so I have to keep warm. I am in my 60's and had severe eczema as a baby. I had dairy and meat for most of my life, but ate very "health conscious" for most of it. 31:48- Betty -- What causes food allergies? How does protein affect the Kidneys? Does the Lymphatic system drain into the venous system, or the kidneys? How do the Adrenal control the Kidneys? Can you get a rash from the fluctuation in blood sugar from eating just fruit? 42:51- Emily -- Please talk about weaning oneself from a pharmaceutical medication for high blood pressure. I take Amlodipine 5MG since November. I have been 80/20 taking an adrenal glandular, kelp and herbal formulas. With all my heart, thank you for your loving kindness, your foreverness. 50:45- Mary -- How can one live on fruit alone, no herbs or greens? Will I become deficient? What is your take on this? 1:00:09- Gabe -- I have a questions regarding fasting. I have tried fruits, berries, and melons for a while now and don't see much sediment in my urine. I don't see much sediment with water fasting for 3 days at a time here and there, but I do see a lot of sediment with dry fasting once a week for 36 hours. Is dry fasting for 36 hours once per week healthy? I also don't understand why I get sediment with dry fasting, but not water fasting? Please let me know what goes on during a dry fast that causes filtering over a water fast. 1:07:47- Shannon -- My husband had a crainionotomy 12 years ago. The last 2 years his head has been pussing and we couldn't find a doctor that would even touch his head. They would just say here are antibiotics and pain meds. The antibiotics never help and he kept telling them that. He gave up and quit going. I found a doctor that sent him to a neurosurgeon and was told he has an infection inside his head. He has a MRI and CAT scan to make sure the infection hasn't reached his skull bone. What herbs can I use to help him with the infection and after surgery for healing? 1:14:42- Claire -- You talk about young souls and old souls. What's the difference and how would a new soul be 'born' or created? And if all matter and atoms are already in existence, what then are souls made of? 1:30:16- Linda/Chuck -- I have been on your herbs for over 4 months now. I started filtering at around 2-3 months. I have bad head and sinus congestion and large polyps. Cold hands and feet and when I drink beer my face gets very pale. What are your thoughts on that? Also, I was using infrared sauna a lot trying to sweat it out. After listening to you I stopped, hopefully not to late. Plus what's your opinion on hot tubs (chlorine) 1:33:39- Sarah -- I am eating a raw food diet, which is predominantly fruit. I eat fruit all day and a salad at night. For my dressing I like to mix tomatoes and avocado, but I am not sure whether half of an avocado a day is too much. I have started including avocado because its winter and a little fat seen to suit me well. I was previously eating only high water content fruits and no salad, but when winter came I began to lose weight so I started including them again. I am not sure whether to continue using avocado because its not very cleansing and its very high in fat, and I do not want to become a high fat raw foodist as it seems to cause a lot of problems for those who attempt it. 1:38:58 - Revolutionman2012 -- My wife suffers from "Depersonalization" I was wondering to hear of any health suggestions you may have for her. We would really appreciate it. We send our love your way. 1:46:22- FlyingWithHorsesUK -- I have a question regarding an equine fatal condition called 'grass sickness' which sadly took the life of my best friend, the majestic white Horse, Joseph, last week. Little is know about this condition, which is most prevalent in the UK. It's thought that a form of ingested neuro-toxin causes gut paralysis, leading to colic & emaciation, and eventually death within a week in 95% of cases. What can we give our horses herbally to help with neuro-toxic reactions, which lead to gut paralysis to help improve recovery? Dr. Morse's website: http://www.DrMorsesHerbalHealthClub.com
Insidermedicine In Depth - April 21, 2010 - Diabetes and Nateglinide
Regularly taking a medication that briefly stimulates the body to produce insulin does not stave off diabetes or cardiovascular disease among high risk individuals with impaired glucose tolerance, according to a trial published in the latest issue of the New England Journal of Medicine. Here is some information on impaired glucose tolerance: • Also known as prediabetes, it is a condition in which the bodys response to insulin is reduced • A sign of impaired glucose tolerance is a blood sugar level that is higher than normal, but not as high as it is among individuals with diabetes • Impaired glucose tolerance is an important risk factor for developing diabetes and cardiovascular disease Researchers representing the NAVIGATOR Study Group randomly assigned over 9,000 individuals with impaired glucose tolerance who also had cardiovascular disease or risk factors for cardiovascular disease to take the drug nateglinide or a placebo up to three times a day. Nateglinide is a short-acting drug, usually taken before a meal, which stimulates the body to produce insulin. After an average follow-up period of five years, about 35% of participants developed diabetes, regardless of whether they were taking the active drug or the placebo. The two groups were also similar with respect to the incidence of several consequences of cardiovascular disease, including heart attacks, strokes, and the need to be hospitalized or undergo treatment for cardiovascular problems. Those taking nateglinide were more likely to experience excessively low blood sugar, however. Todays research suggests that short-acting drugs that help stimulate insulin production do not offer health benefits for high risk individuals who have impaired glucose tolerance.
Views: 352 insidermedicine